Host Immune Responses and Pathogenesis to Brucella spp. Infection

doi:10.3390/books978-3-0365-2419-1

Download Url(s)

https://mdpi.com/books/pdfview/book/4567

Contributor(s)

Oliveira, Sergio Costa (editor)

Giambartolomei, Guillermo (editor)

Language

English

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Abstract

Brucellosis, caused by the facultative intracellular bacteria Brucella species, is one the most prevalent zoonosis worldwide. • The articles described in this book report several aspects of host-Brucella interactions. • The findings described here will help to advance in the comprehension of bacterial pathogenesis and contribute to the future development of drugs or vaccines to control brucellosis.

URI

https://directory.doabooks.org/handle/20.500.12854/76974

Keywords

Recombinant vaccine; divalent vaccine; brucellosis; Omp25; L7/L12; Brucella abortus 544; ST2 receptor; Brucella abortus; oral infection; human endometrial cells; internalization; intracellular replication; decidualization; chemokines; macrophages; Brucella; HSC; MHC; IL-10; cell cycle; (p)ppGpp; rsh; neurobrucellosis; platelets; brain microvascular endothelial cells; endothelial cells; adhesins; Ig-like domain; monomeric autotransporters; trimeric autotransporters; extracellular matrix; polar localization; virulence factors; vaccine candidates; fibronectin; canonical inflammasome; non-canonical inflammasome; NLR; pyroptosis; ASC; caspase-11; caspase-1; IL-1β; gDNA; replication niche; reservoir; persistence; survival; chronic infection; n/a