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dc.contributor.editorAhmad, Rizwan
dc.date.accessioned2024-03-07T17:16:54Z
dc.date.available2024-03-07T17:16:54Z
dc.date.issued2024
dc.identifierONIX_20240307_9781837682539_288
dc.identifier.issn2632-0983
dc.identifier.urihttps://directory.doabooks.org/handle/20.500.12854/135379
dc.description.abstractReactive Oxygen Species (ROS) are molecules generated naturally during cellular metabolic processes. They act as signaling agents that oversee specific biochemical pathways, playing a vital role in cell function and survival. However, an imbalance in ROS signaling or excessive ROS production can have harmful effects on the pathophysiology of diseases. ROS are crucial to cell signaling and are involved in various physiological processes. They modulate gene expression, regulate cell cycle progression, and influence immune responses. Although ROS are essential for normal cellular functions, an overabundance of these molecules can lead to oxidative stress, causing DNA damage, lipid peroxidation, and protein oxidation, adversely impacting cell function and leading to various diseases. Therefore, it is critical to regulate ROS levels precisely to maintain cellular homeostasis. Reactive Oxygen Species - Advances and Developments is a comprehensive book that delves into the intricacies of ROS. It provides invaluable insights to researchers in the field, equipping them with the essential tools and knowledge to advance their work in this critical area, leading to the development of novel therapeutic interventions to manage various illnesses.
dc.languageEnglish
dc.relation.ispartofseriesBiochemistry
dc.subject.classificationthema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSF Cellular biology (cytology)en_US
dc.titleReactive Oxygen Species
dc.title.alternativeAdvances and Developments
dc.typebook
oapen.identifier.doi10.5772/intechopen.107774
oapen.relation.isPublishedBy78a36484-2c0c-47cb-ad67-2b9f5cd4a8f6
oapen.relation.isbn9781837682539
oapen.relation.isbn9781837682096
oapen.relation.isbn9781837682546
oapen.imprintIntechOpen
oapen.series.number50
oapen.pages174


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